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The mechanisms governing non-recurrent human structural variation SV are diverse and often poorly understood. I am investigating how human DNA maintains fidelity in the context of a repetitive genome. Human Alu elements number over one million copies per human genome, and karaca chester yeşil studies have found that these repeat sequences often mediate SVs in some loci.

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Karaca chester yeşil

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Balachandran, Parithi; Beck, Christine R Chromosome research : an international journal on the molecular, supramolecular and evolutionary aspects of chromosome biology Jan. Christine R. Integration of karaca chester yeşil sequencing and genomic analyses reveals potential Smith-Magenis-like causative genes.

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Karaca chester yeşil

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Lupski, M. Beck, Ph. Through computational, molecular biological and genomic techniques, we will identify regions susceptible to this form of SV and investigate the enzymes that limit or promote Alu-mediated rearrangements. Journal Articles Small polymorphisms are a source of ancestral bias in structural variant breakpoint placement. Balachandran, Parithi; Beck, Christine R Chromosome research : an international journal on the molecular, supramolecular and evolutionary aspects of chromosome biology Jan;. Christine R. Beck, Ph. Journal Articles Small polymorphisms are a source of ancestral bias in structural variant breakpoint placement. Website s :. Iowa State University Biochemistry Ph. James R. The mechanisms governing non-recurrent human structural variation SV are diverse and often poorly understood.

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Invited Speaker. Human Alu elements number over one million copies per human genome, and recent studies have found that these repeat sequences often mediate SVs in some loci. James R. Lupski, M. Research Fellow, Laboratory of Dr. Whole-exome sequencing links TMCO1 defect syndrome with cerebro-facio-thoracic dysplasia. Integration of exome sequencing and genomic analyses reveals potential Smith-Magenis-like causative genes. Human Alu elements number over one million copies per human genome, and recent studies have found that these repeat sequences often mediate SVs in some loci. Integration of exome sequencing and genomic analyses reveals potential Smith-Magenis-like causative genes. Website s :. Balachandran, Parithi; Beck, Christine R Chromosome research : an international journal on the molecular, supramolecular and evolutionary aspects of chromosome biology Jan;. Beck, Ph.